• Distributes blood to specific organs • Little to no elastic tissue • Brachial, femoral, renal, and splenic • Smooth muscle layers ¾ wall thicknes
Q.9.
Vasomotion
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
- by relaxation of the smooth muscl
0%
-chronic low resting BP -Caused by blood loss, dehydration, anemia
0%
is a quick and powerful way of altering blood pressure and flow
Q.10.
Interstitial hydrostatic pressure
0%
Arterial end = +3 out Venous end = +3 out
0%
Artery flows directly into vein bypassing capillaries
0%
Arterial end = -8 outVenous end = -8 out
0%
Arterial end = 20 inVenous end = 20 in
Q.11.
drain blood from brain to
0%
internal jugular vein
0%
subclavian vein
0%
Arterial end = 20 inVenous end = 20 in
0%
aorta and carotid arteries.
Q.12.
Blood Pressure
0%
-Normal value, young adult: 120/75 mm Hg -Blood pressure (BP) - the force that blood exerts against a vessel wal-Sphygmomanometer and Brachial artery -Pulse pressure
0%
-Weak point in an artery or the heart wall-Most common sites: abdominal aorta, renal arteries, and arterial circle at the base of the brain
0%
minimum arterial BP taken during ventricular relaxation (diastole) between heart beats
0%
peak arterial BP taken during ventricular contraction (ventricular systole)
Q.13.
The fluids leave the capillaries at the arterial end because?
0%
celiac trunk, superior mesenteric artery, renal arteries, gonadal arteries, inferior mesenteric artery, and common iliac arteries.
0%
D. Central venous pressure being greater than pressure in the venules
0%
the net filtration pressure of the blood is higher at the arterial end than it is at the venous end.
0%
The opposition to flow that blood encounters in vessels away from the heart
Q.14.
The most important force driving reabsorption at the venous end of a capillary is
0%
blood colloid osmotic pressure.
0%
C. increased blood pressure
0%
draws fluid into capillary
0%
the great saphenous vein
Q.15.
Blood flow to the _______________ remains quite stable even when mean arterial pressure (MAP) fluctuates from 60 to 140 mm Hg.
0%
considerably lower
0%
hypothalamus
0%
the vagus nerve.
0%
the great saphenous vein
Q.16.
Three primary causes of Edema
0%
occurs when cardiac output is low because too little blood is returning to the heart.
0%
C. increased blood pressure
0%
- smallest arteries • Control amount of blood to various organs* - are most significant point of control over peripheral resistance and flow -Arterioles produce half of the total peripheral resistance
the heart beats faster and harder, increasing cardiac output and blood pressure.
0%
D. Central venous pressure being greater than pressure in the venules
Q.22.
Arterial anastomosis
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
- Two arteries merge - Collateral (alternative) routes of blood supply - Coronary circulation and around joints
0%
- Increased CO and BP - Increased respiratory rate (thoracic pump) - Increased skeletal muscle pump
0%
peak arterial BP taken during ventricular contraction (ventricular systole)
Q.23.
This is the longest vein, and portions of this vein are commonly used as grafts in coronary bypass surgery.
0%
the vagus and glossopharyngeal nerves.
0%
the vagus nerve.
0%
the great saphenous vein
0%
The first, second, and third choices are correct
Q.24.
Peripheral Resistance
0%
the heart beats faster and harder, increasing cardiac output and blood pressure.
0%
peak arterial BP taken during ventricular contraction (ventricular systole)
0%
The opposition to flow that blood encounters in vessels away from the heart
0%
-Tissue necrosis, pulmonary and cerebral edema, circulatory shock
Q.25.
Neurogenic shock
0%
- Most common - One vein empties directly into another - Vein blockage less serious
0%
-chronic low resting BP -Caused by blood loss, dehydration, anemia
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
is a form of venous pooling shock that results from a sudden loss of vasomotor tone, allowing the vessels to dilate.
Q.26.
Vessel changes names as passes to different regions
0%
Arterial end = -8 outVenous end = -8 out
0%
Subclavian to axillary to brachial to radial and ulnar
0%
subclavian vein
0%
D. Central venous pressure being greater than pressure in the venules
Q.27.
Arteriovenous anastomosis (shunt)
0%
Artery flows directly into vein bypassing capillaries
0%
Arterial end = 20 inVenous end = 20 in
0%
Arterial end = +3 out Venous end = +3 out
0%
Arterial end = -8 outVenous end = -8 out
Q.28.
Veins
0%
Greater capacity for blood than arterie
0%
receives most of the blood from the brain
0%
Arterial end = -8 outVenous end = -8 out
0%
wrap around capillaries and contract, regulating blood flow
Q.29.
Approximately what percent of fluid that exits the capillaries at the arterial end renters the capillaries at the venous end?
0%
90%
0%
tunica externa; vasa vasorum
0%
blood pressure.
0%
hypothalamus
Q.30.
Hypovolemic shock
0%
high blood pressure Chronic is resting BP > 140/90 Can weaken small arteries and cause aneurysms
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
is a form of venous pooling shock that results from a sudden loss of vasomotor tone, allowing the vessels to dilate.
0%
occurs when bacterial toxins trigger vasodilation and increased capillary permeability.
Q.31.
Net filtration or reabsorption pressure
0%
Arterial end = -8 outVenous end = -8 out
0%
Arterial end = 28 in Venous end = 28 in
0%
Arterial end = 13 outVenous end = 7 in
0%
Arterial end = 30 out Venous end = 10 out
Q.32.
Colloid osmotic pressures (COP) or oncotic pressure
0%
blood colloid osmotic pressure.
0%
drives fluid out of capillary (major force)
0%
Arterial end = 28 in Venous end = 28 in
0%
draws fluid into capillary
Q.33.
Internal carotid artery (ICA)
0%
Arterial end = 28 in Venous end = 28 in
0%
Arterial end = +3 out Venous end = +3 out
0%
supplies most external head structures
0%
supplies 80% of the cerebrum
Q.34.
Colloid osmotic pressures (COP) Tissue fluid
0%
aorta and carotid arteries.
0%
the vagus and glossopharyngeal nerves.
0%
drain the external structures of the head
0%
Arterial end = -8 outVenous end = -8 out
Q.35.
Oncotic pressure (net COP)
0%
Arterial end = -8 outVenous end = -8 out
0%
peak arterial BP taken during ventricular contraction (ventricular systole)
0%
Arterial end = +3 out Venous end = +3 out
0%
Arterial end = 20 inVenous end = 20 in
Q.36.
From superior to inferior, the major branches of the abdominal aorta are
0%
• Biggest arteries • Aorta, common carotid, subclavian, pulmonary trunk, and common iliac arteries • Much elastic tissue • Greatest pulse pressure
0%
-Normal value, young adult: 120/75 mm Hg -Blood pressure (BP) - the force that blood exerts against a vessel wal-Sphygmomanometer and Brachial artery -Pulse pressure
0%
- Raise or lower BP throughout the whole body - Re-routing blood from one region to another for perfusion of individual organs (Rest vs. Exercise
0%
celiac trunk, superior mesenteric artery, renal arteries, gonadal arteries, inferior mesenteric artery, and common iliac arteries.
Q.37.
Colloid osmotic pressures (COP) Blood
0%
the vagus and glossopharyngeal nerves.
0%
Artery flows directly into vein bypassing capillaries
0%
Arterial end = 28 in Venous end = 28 in
0%
Arterial end = 20 inVenous end = 20 in
Q.38.
Upper limb is drained by
0%
medulla oblongata.
0%
veins
0%
subclavian vein
0%
supplies 80% of the cerebrum
Q.39.
Systolic pressure
0%
peak arterial BP taken during ventricular contraction (ventricular systole)
0%
minimum arterial BP taken during ventricular relaxation (diastole) between heart beats
0%
link arterioles to capillaries
0%
- Two arteries merge - Collateral (alternative) routes of blood supply - Coronary circulation and around joints
Q.40.
Arterioles
0%
- Raise or lower BP throughout the whole body - Re-routing blood from one region to another for perfusion of individual organs (Rest vs. Exercise
0%
- smallest arteries • Control amount of blood to various organs* - are most significant point of control over peripheral resistance and flow -Arterioles produce half of the total peripheral resistance
0%
• Biggest arteries • Aorta, common carotid, subclavian, pulmonary trunk, and common iliac arteries • Much elastic tissue • Greatest pulse pressure
- Blood viscosity • Anemia and hypoproteinemia speed flow • Polycythemia and dehydration slow flow - Vessel length • The farther the liquid travels through a tube = more friction • Pressure and flow decline with distance - Vessel radius - most powerful influence over flow • Only significant way of controlling peripheral resistance. • Vasomotion
0%
-Tissue necrosis, pulmonary and cerebral edema, circulatory shock
0%
draws fluid into capillary
0%
the net filtration pressure of the blood is higher at the arterial end than it is at the venous end.
Q.43.
Anaphylactic shock
0%
is caused by inadequate pumping by the heart, usually as a result of myocardial infarction.
0%
occurs when bacterial toxins trigger vasodilation and increased capillary permeability.
0%
results from exposure to an antigen to which a person is allergic, such as bee venom.
0%
-Tissue necrosis, pulmonary and cerebral edema, circulatory shock
Q.44.
Metarterioles
0%
- Two arteries merge - Collateral (alternative) routes of blood supply - Coronary circulation and around joints
0%
link arterioles to capillaries
0%
the point where two blood vessels merge
0%
failure of venous valves
Q.45.
Diastolic pressure
0%
is caused by inadequate pumping by the heart, usually as a result of myocardial infarction.
0%
results from exposure to an antigen to which a person is allergic, such as bee venom.
0%
minimum arterial BP taken during ventricular relaxation (diastole) between heart beats
0%
- Two arteries merge - Collateral (alternative) routes of blood supply - Coronary circulation and around joints
Q.46.
Low Venous Return (LVR) Shock
0%
- Increased CO and BP - Increased respiratory rate (thoracic pump) - Increased skeletal muscle pump
0%
Artery flows directly into vein bypassing capillaries
0%
is caused by inadequate pumping by the heart, usually as a result of myocardial infarction.
0%
occurs when cardiac output is low because too little blood is returning to the heart.
Q.47.
- Vasodilation
0%
is a quick and powerful way of altering blood pressure and flow
0%
link arterioles to capillaries
0%
passes between clavicle and 1st rib
0%
- by relaxation of the smooth muscl
Q.48.
The action potential from the brain to the heart travels along...
0%
the vagus and glossopharyngeal nerves.
0%
subclavian vein
0%
the vagus nerve.
0%
blood colloid osmotic pressure.
Q.49.
Hypertension
0%
wrap around capillaries and contract, regulating blood flow
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
is a quick and powerful way of altering blood pressure and flow
0%
high blood pressure Chronic is resting BP > 140/90 Can weaken small arteries and cause aneurysms
Q.50.
Three ways of controlling vasomotion
0%
D. Central venous pressure being greater than pressure in the venules
0%
1. Local control 2. Neural control 3. Hormonal control
0%
Arterial end = 13 outVenous end = 7 in
0%
drain the external structures of the head
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